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Support counts the member genes carrying the term. % of genes is that count over all 52 members — the strict reading of “the whole family agrees”. % annotated is the same count over only those members for which this database has a prediction of that type, which shows how uniform the evidence is where evidence exists.
| Source | Term | Name / description | Support | % of genes | % annotated | Consistency |
|---|---|---|---|---|---|---|
| Pfam | PF00001 | 7tm_1 — 7 transmembrane receptor (rhodopsin family) | 47 / 52 | 90.4% | 97.9% of 48 | ≥80% support |
| GO | GO:0004930 Molecular Function | G protein-coupled receptor activity | 47 / 52 | 90.4% | 97.9% of 48 | ≥80% support |
| GO | GO:0007186 Biological Process | G protein-coupled receptor signaling pathway | 47 / 52 | 90.4% | 97.9% of 48 | ≥80% support |
| GO | GO:0016020 Cellular Component | membrane | 47 / 52 | 90.4% | 97.9% of 48 | ≥80% support |
| PANTHER | PTHR24372 | GLYCOPROTEIN HORMONE RECEPTOR | 28 / 52 | 53.9% | 58.3% of 48 | ≥50% support |
| GO | GO:0005887 Cellular Component | plasma membrane | 35 / 52 | 67.3% | 72.9% of 48 | ≥50% support |
| GO | GO:0005886 Cellular Component | plasma membrane | 28 / 52 | 53.9% | 58.3% of 48 | ≥50% support |
| GO | GO:0007189 Biological Process | adenylate cyclase-activating G protein-coupled receptor signaling pathway | 28 / 52 | 53.9% | 58.3% of 48 | ≥50% support |
| GO | GO:0007190 Biological Process | activation of adenylate cyclase activity | 28 / 52 | 53.9% | 58.3% of 48 | ≥50% support |
| GO | GO:0008528 Molecular Function | G protein-coupled peptide receptor activity | 28 / 52 | 53.9% | 58.3% of 48 | ≥50% support |
| GO | GO:0009755 Biological Process | hormone-mediated signaling pathway | 28 / 52 | 53.9% | 58.3% of 48 | ≥50% support |
Column guide: Top NCBI-NR hit and Top UniProt hit are the closest characterised sequences found by homology search — they are not identifiers of the CnidoSite gene itself. Where a species has no Swiss-Prot hit above threshold the UniProt column is shown as –. Click a gene ID for its full annotation page.
